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Vitrupath

Welcome to Vitrupath

A New Foundation

10,000 years of civilizations, billions of instances of individual Human experiences and events, interactive with creative forces of the Universes, derivatizing achievement of the nuances of favor intended and possible for Humanity, a new perspective of everything. 

S-adenosyl methylene cysteine( S-adenosyl Hcy) < 0.012 µm/L, methylene cysteine (Hcy) < 6 or 7 µm/L statistically toward 3.7 µm/L as a therapeutic objective, tmao intervened during each intervention and proactively = 99.9+ percent decrease in risk for all cause abated being and morbidities 

Introduction
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Hydrogen Anion

Hydrogen Anion as high energy electrons in carriers of H2e1p, eV energy, fluorescent energy, wave function and superposition maintain a foundational biological alkaline disequilibrium as a an alkaline  background pH which compares with foreground H+ variants which is used for synthesis of energy, polymerization and exhibition of self determinism by living things.  CH3, methyl group, exhibits to hydrogens and a hydrogen anion, and the enzyme PEMT packs CH3 into membranes. Oxygen is essential to the synthesis of ATP by the thioretinaco ozonide complex when it becomes the complex 5 ATP synthase complex.  Disruption of this complex or commandeering aspects of its supply pathway are canonical aspects of disease.  Counteracting methylene cysteine (clinically Hcy) sequestration of electrons from tissue, structure, membranes, hydrogen anion and carriers of  hydrogen prevents protonation which disrupts strength of optimal background pH.  How to counteract these?

Pemt Enzyme

Pemt1 emerges at conception in the endoplasmic reticulum while Pemt2 and Pemt3 emerge at conclusion of gestation only in the membrane shared by the mitochondria and the Endoplasmic Reticulum to regulate Pemt1.   Pemt1 produces brand new choline as phosphatidylcholine with healing fatty acids, renews structure, packs hydrogen anion into membranes, produces super clot busters, deteriorates the structure of oncology causing molecules,  counteracts growth hormones in membranes, cause serine protease activity to promote plasticity levels exhibited otherwise in gestational development, decreases methylene cysteine,  deteriorates microplastics in tissues using phosphatidylcholine, and promotes expression of oxytocin which is a foundation for human relationships, learning, compassion, neurological basis of social behavior and foundation for civilizations.  How to counteract these?

Counteract Methylene Cysteine and tmao

This review found that the average typical methylene cysteine by age and changes by age were nearly precisely positive correlate with gompertz makeham sigmoidal graph used by indemnification industries to ascertain risk for no longer exhibiting vital being. Also found was a study and correlated data that shows risk for abated vital being from any cause over a decade of observation as being about 500 when methylene cysteine (clinically known as Hcy) was statistically above 6 or 7 µm/L while while below 6 or 7  6 or 7 µm/L toward 3.7 um/L as a clinical objective the risk for abated being over a decade for all cause was only 1.   Tmao is either the or among the most correlated causes of sudden abated being, sudden diminished outcomes an perioperative complications. Counteracting these can massively decrease risk by over 99 percent. Methylene cysteine

sequesters electrons. How to counteract these?

Environment Protection Recommndation

 Exposure to EMF causes particular factors to be expressed and causes conditions essential to disease and dysbiosis.  EMF protection capabilities, kits, and capabilities for dwelling, building, physiology, cognition and therapy are available.  Environmental particulate, exposure to toxins, and even particulate in nutritional factors, along with diminished health status cause HIF hypoxia factor, the aryl hydrogen carbon receptor, and the HIF aryl hydrogen carbon receptor to be expressed and change the ability of programmed function of tissue to occur accurately.  Choline Kinase alpha also is increased in disease to increase resiliency of tissue ephemerally while causing disease when non ephemerally or overactivated. Diseases, thus, sequester hydrogen anion, disrupt thioretinaco ozonide supply and activity, commandeer and divert ATP, prevent  programmed or causes tissue deterioration, change oxygenation, and deterioration ATP use to produce S-adenosyl methionine which is the primary substrate for the enzyme Pemt. There are numerous more intricate factors in disease and capabilities which have the potential or preventing. improving and counteracting every disease and diminished outcome.   How to counteract these? 

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